Pestilential Pollutants
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Pestilential Pollutants
Natural occurring phenomenons occur every day in every society. A major event that most people dont think a lot about it is both air pollution and water pollution. Air pollution is defined as chemicals and gases in our air that are not a part of Earths air or are not normal in these amounts, and water pollution refers to the addition of foreign substances (pollutants) to a water source. Air pollution affects humans and land animals, while water pollutants affect ocean life. There is no way to physically stop pollutants from being put into our air and water, however it is possible to cut back on the amount of exposure our nation has. Pollution is a global killer. There are many studies and research projects constructed to predict and understand what harm pollutants cause to our nation as a whole. Two separate articles discuss the cause of asthma, diabetes, obesity, and the oxidative-stress gene in people due to traffic-related air pollution. Traffic-related pollution is mainly emitted through the exhaust of motor vehicles and the combustion of fossil fuels and oils. While these articles are both discussing similar healthcare risks, they are also every contradictory in other physical ways.
Traffic-related air pollution is very hazardous, however people never wake up thinking when they drive to work or school that day that they are harming numerous lives around them. Article one titled “Traffic-Related Air Pollution, Oxidative Stress Genes, and Asthma (ECHRS)” discusses the major risks and causes of asthma in children and female adults. It also explains how oxidative stress triggers inflammatory response and cytokine production. Scientists believe that genetic variants involved in inflammation and protection against reactive oxygen species may influence the response to air pollutants, which means the oxidative stress gene consists of components that react negatively with the pollution in the air. Also, article one enforces the evidence that gene-air pollution interactions on asthma have been reported only in children. This doesnt mean that asthma only affects and develops in children; it just means that air pollution in early life may biologically differ from adult-hood. Research from the European Community Respiratory Health Survey (ECRHS) stated that traffic-related pollution was positively associated with the new-onset of asthma in adults. A study was taken place to identify interactions between genes and traffic-related pollution on asthma, which in conclusion led to the assumption that women are at higher risks to become diagnosed with asthma triggered by traffic-related pollution than men.
Article two focused on the characteristics of men that may have been triggered by traffic-related pollution, for example: diabetes, obesity, and the oxidative stress gene. “Traffic-Related Air Pollution and QT Interval: Modification by Diabetes, Obesity, and Oxidative Stress Gene Polymorphisms in the Normative Aging Study” uses evidence from epidemiologic studies that shows relations between increased ambient air pollution and increased daily hospital admission and premature death. This articles focuses mainly on more serious, life threatening